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tacrolimus (tac) enzyme-linked immunoassay (elisa) kits  (MyBiosource Biotechnology)

 
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    MyBiosource Biotechnology tacrolimus (tac) enzyme-linked immunoassay (elisa) kits
    Tacrolimus (Tac) Enzyme Linked Immunoassay (Elisa) Kits, supplied by MyBiosource Biotechnology, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/tacrolimus (tac) enzyme-linked immunoassay (elisa) kits/product/MyBiosource Biotechnology
    Average 90 stars, based on 1 article reviews
    tacrolimus (tac) enzyme-linked immunoassay (elisa) kits - by Bioz Stars, 2026-02
    90/100 stars

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    Longitudinal changes in oxidative stress biomarkers <t>(TAC,</t> <t>SOD,</t> CAT, GSH, LPO) across four timepoints in the intervention and control groups. Changes in oxidative stress biomarkers between the intervention group (IG) and control group (CG) over the study period (t 0 to t 3 ): (A) Plasma Total Antioxidant Capacity (TAC), (B) Superoxide Dismutase (SOD), (C) Catalase (CAT), (D) Glutathione (GSH), and (E) Lipid Peroxidation (LPO). Data are expressed as mean ±2 standard error (SE). Statistical analysis was conducted using two-way repeated measures ANOVA with Bonferroni-adjusted post hoc comparisons. Asterisks indicate significant between-group differences at individual timepoints (* P < 0.05, ** P < 0.01, *** P < 0.001). (A) Total Antioxidant Capacity (TAC) (B) Superoxide Dismutase (SOD) (C) Catalase Enzyme (CAT) (D) Glutathione (GSH) (E) Lipid peroxidation (LPO).
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    Image Search Results


    AF impairs spheroid formation and growth by inducing apoptotic cell death and ROS production. (A) Percentage of apoptotic and necrotic cells shown by flow cytometry analysis of annexin V/propidium iodide‐stained. Spheroids were grown for 72 h with increasing AF concentrations. Flow cytometric images are representative of three independent experiments. Histograms report the mean values ± SD. (B) ROS production during spheroid formation and growth was evaluated using the DCFDA probe. Histograms represent the percentage of P2 (ROS‐positive) cells. (C) Total antioxidant capacity (ORAC assay). Histogram reports the mean values ± SD of three independent experiments. * p < 0.05, ** p < 0.01, *** p < 0.001.

    Journal: Journal of Cellular and Molecular Medicine

    Article Title: Assessing Auranofin for Second‐Line Use in Chemoresistant Ovarian Cancer: Effects on Tumour Spheroid and Primary Cell Growth

    doi: 10.1111/jcmm.70681

    Figure Lengend Snippet: AF impairs spheroid formation and growth by inducing apoptotic cell death and ROS production. (A) Percentage of apoptotic and necrotic cells shown by flow cytometry analysis of annexin V/propidium iodide‐stained. Spheroids were grown for 72 h with increasing AF concentrations. Flow cytometric images are representative of three independent experiments. Histograms report the mean values ± SD. (B) ROS production during spheroid formation and growth was evaluated using the DCFDA probe. Histograms represent the percentage of P2 (ROS‐positive) cells. (C) Total antioxidant capacity (ORAC assay). Histogram reports the mean values ± SD of three independent experiments. * p < 0.05, ** p < 0.01, *** p < 0.001.

    Article Snippet: The evaluation of cell death was carried out using the TACS annexin V/propidium iodide staining kit (Trevigen, Gaithersburg, MD, USA) according to the manufacturer's instructions.

    Techniques: Flow Cytometry, Staining, ORAC Assay

    AF induces necrotic and apoptotic cell death in pre‐formed spheroids. Percentage of apoptotic and necrotic cells shown by flow cytometry analysis of annexin V/propidium iodide‐stained. Spheroids were treated for 72 h with AF concentrations corresponding to IC50 values. Flow cytometric images are representative of three independent experiments. Histograms report the mean values ± SD. * p < 0.05, ** p < 0.01.

    Journal: Journal of Cellular and Molecular Medicine

    Article Title: Assessing Auranofin for Second‐Line Use in Chemoresistant Ovarian Cancer: Effects on Tumour Spheroid and Primary Cell Growth

    doi: 10.1111/jcmm.70681

    Figure Lengend Snippet: AF induces necrotic and apoptotic cell death in pre‐formed spheroids. Percentage of apoptotic and necrotic cells shown by flow cytometry analysis of annexin V/propidium iodide‐stained. Spheroids were treated for 72 h with AF concentrations corresponding to IC50 values. Flow cytometric images are representative of three independent experiments. Histograms report the mean values ± SD. * p < 0.05, ** p < 0.01.

    Article Snippet: The evaluation of cell death was carried out using the TACS annexin V/propidium iodide staining kit (Trevigen, Gaithersburg, MD, USA) according to the manufacturer's instructions.

    Techniques: Flow Cytometry, Staining

    Longitudinal changes in oxidative stress biomarkers (TAC, SOD, CAT, GSH, LPO) across four timepoints in the intervention and control groups. Changes in oxidative stress biomarkers between the intervention group (IG) and control group (CG) over the study period (t 0 to t 3 ): (A) Plasma Total Antioxidant Capacity (TAC), (B) Superoxide Dismutase (SOD), (C) Catalase (CAT), (D) Glutathione (GSH), and (E) Lipid Peroxidation (LPO). Data are expressed as mean ±2 standard error (SE). Statistical analysis was conducted using two-way repeated measures ANOVA with Bonferroni-adjusted post hoc comparisons. Asterisks indicate significant between-group differences at individual timepoints (* P < 0.05, ** P < 0.01, *** P < 0.001). (A) Total Antioxidant Capacity (TAC) (B) Superoxide Dismutase (SOD) (C) Catalase Enzyme (CAT) (D) Glutathione (GSH) (E) Lipid peroxidation (LPO).

    Journal: American Journal of Lifestyle Medicine

    Article Title: Lifestyle Medicine as a Pathway to Longevity: The Impact of the Healthy Lifestyle Community Program (Cohort 2, HLCP-2) on Oxidative Stress Biomarkers in the Adult Rural German Population

    doi: 10.1177/15598276251348197

    Figure Lengend Snippet: Longitudinal changes in oxidative stress biomarkers (TAC, SOD, CAT, GSH, LPO) across four timepoints in the intervention and control groups. Changes in oxidative stress biomarkers between the intervention group (IG) and control group (CG) over the study period (t 0 to t 3 ): (A) Plasma Total Antioxidant Capacity (TAC), (B) Superoxide Dismutase (SOD), (C) Catalase (CAT), (D) Glutathione (GSH), and (E) Lipid Peroxidation (LPO). Data are expressed as mean ±2 standard error (SE). Statistical analysis was conducted using two-way repeated measures ANOVA with Bonferroni-adjusted post hoc comparisons. Asterisks indicate significant between-group differences at individual timepoints (* P < 0.05, ** P < 0.01, *** P < 0.001). (A) Total Antioxidant Capacity (TAC) (B) Superoxide Dismutase (SOD) (C) Catalase Enzyme (CAT) (D) Glutathione (GSH) (E) Lipid peroxidation (LPO).

    Article Snippet: TAC, SOD, CAT, GSH activities and LPO level were detected using commercially available kits from Cayman Chemicals, Ann Arbor, MI, USA according to the manufacturer’s instructions.

    Techniques: Control, Clinical Proteomics

    Gender-based analysis of oxidative stress biomarkers pre- and post-intervention. (A) Total antioxidant capacity (TAC mg/mL). (B) Superoxide dismutase (SOD U/mL). (C) Catalase enzyme activity (CAT U/mL). (D) Glutathione (GSH μmol/L). (E) Lipid peroxidation (LPO μmol/L). Illustrates the gender-specific impact of the HPCL-2 on oxidative stress biomarkers after 10 weeks within the intervention group. The analysis was conducted within the intervention group, evaluating biomarkers including: (a) total antioxidant capacity (TAC); (b) superoxide dismutase (SOD); (c) catalase enzyme (CAT); (d) glutathione (GSH), and (e) lipid peroxidation (LPO). The comparison was made between male (n = 32) and female (n = 67) participants to discern variations in biomarker levels after the intervention. Each bar on the graph represents the mean change in biomarker levels, with error bars indicating standard deviations and significance levels set at P < 0.05. Notably, a significant gender-specific difference was observed solely in the catalase enzyme ( P = 0.009), while TAC ( P = 0.97), SOD ( P = 0.23), GSH ( P = 0.75), and LPO ( P = 0.74) did not exhibit statistically significant gender differences ( P > 0.05) after the 10-week intervention within the intervention group.

    Journal: American Journal of Lifestyle Medicine

    Article Title: Lifestyle Medicine as a Pathway to Longevity: The Impact of the Healthy Lifestyle Community Program (Cohort 2, HLCP-2) on Oxidative Stress Biomarkers in the Adult Rural German Population

    doi: 10.1177/15598276251348197

    Figure Lengend Snippet: Gender-based analysis of oxidative stress biomarkers pre- and post-intervention. (A) Total antioxidant capacity (TAC mg/mL). (B) Superoxide dismutase (SOD U/mL). (C) Catalase enzyme activity (CAT U/mL). (D) Glutathione (GSH μmol/L). (E) Lipid peroxidation (LPO μmol/L). Illustrates the gender-specific impact of the HPCL-2 on oxidative stress biomarkers after 10 weeks within the intervention group. The analysis was conducted within the intervention group, evaluating biomarkers including: (a) total antioxidant capacity (TAC); (b) superoxide dismutase (SOD); (c) catalase enzyme (CAT); (d) glutathione (GSH), and (e) lipid peroxidation (LPO). The comparison was made between male (n = 32) and female (n = 67) participants to discern variations in biomarker levels after the intervention. Each bar on the graph represents the mean change in biomarker levels, with error bars indicating standard deviations and significance levels set at P < 0.05. Notably, a significant gender-specific difference was observed solely in the catalase enzyme ( P = 0.009), while TAC ( P = 0.97), SOD ( P = 0.23), GSH ( P = 0.75), and LPO ( P = 0.74) did not exhibit statistically significant gender differences ( P > 0.05) after the 10-week intervention within the intervention group.

    Article Snippet: TAC, SOD, CAT, GSH activities and LPO level were detected using commercially available kits from Cayman Chemicals, Ann Arbor, MI, USA according to the manufacturer’s instructions.

    Techniques: Activity Assay, Comparison, Biomarker Discovery